Tải bản đầy đủ (.pdf) (3 trang)

Andersons pediatric cardiology 187

Bạn đang xem bản rút gọn của tài liệu. Xem và tải ngay bản đầy đủ của tài liệu tại đây (60.51 KB, 3 trang )

causingimpedimenttoleftventricularinflow.
UltrasoundObstetGynecol.2015;45(6):664–
669.
103.SzwastA,PuttM,GaynorJW,LichtD,Rychik
J.Cerebrovascularresponsetomaternal
hyperoxygenation(MH)infetuseswith
hypoplasticleftheartsyndrome(HLHS)
dependsupongestationalage(GA)andbaseline
cerebrovascularresistance.UltrasoundObstet
Gynecol.2017.
104.KohlT.Chronicintermittentmaterno-fetal
hyperoxygenationinlategestationmayimprove
onhypoplasticcardiovascularstructures
associatedwithcardiacmalformationsinhuman
fetuses.PediatrCardiol.2010;31(2):250–263.
105.ZengS,ZhouJ,PengQ,etal.Sustained
maternalhyperoxygenationimprovesaorticarch
dimensionsinfetuseswithcoarctation.SciRep.
2016;6:39304.
106.BaschatAA,GembruchU,ReissI,GortnerL,
DiedrichK.Demonstrationoffetalcoronary
bloodflowbydopplerultrasoundinrelationto
arterialandvenousflowvelocitywaveformsand
perinataloutcome-the‘heart-sparingeffect.
UltrasoundObstetGynecol.1997;9:162–172.
107.GembruchU,BaschatAA.Demonstrationof
fetalcoronarybloodflowbycolour-codedand


pulsedwavedopplersonography:apossible
indicatorofseverecompromiseandimpending


demiseinintrauterinegrowthretardation.
UltrasoundObstetGynecol.1996;7:10–16.
108.RellerMD,BursonMA,LohrJL,MortonMJ,
ThornburgKL.Nitricoxideisanimportant
determinantofcoronaryflowatrestandduring
hypoxemicstressinfetallambs.AmJPhysiol.
1995;269:H2074–H2081.
109.ChaouiR.Thefetal‘heart-sparingeffect’
detectedbytheassessmentofcoronaryblood
flow:afurtherominoussignoffetal
compromise.UltrasoundObstetGynecol.
1996;7:5–9.
110.BaschatAA,GembruchU,ReissI,etal.
Relationshipbetweenarterialandvenous
dopplerandperinataloutcomeinfetalgrowth
restriction.UltrasoundObstetGynecol.
2000;16:407–413.
111.WohlmuthC,BoudreauxD,MoiseKJJr,etal.
Cardiacpathophysiologyintwin-twin
transfusionsyndrome:newinsightsintoits
evolution.UltrasoundObstetGynecol.2017.
112.LauVK,SagawaK,SugaH.Instantaneous
pressure-volumerelationshipofrightatrium
duringisovolumiccontractionincanineheart.
AmJPhysiol.1979;236:H672–H679.


113.ClarkEB,HuN,DummettJL,etal.Ventricular
functionandmorphologyinchickembryofrom
stages18to29.AmJPhysiol.1986;250:H407–

H413.
114.HeneinMY,GibsonDG.Normallongaxis
function.Heart.1999;81(2):111–113.
115.MäkikallioK,JouppilaP,RäsänenJ.Human
fetalcardiacfunctionduringthefirsttrimesterof
pregnancy.Heart.2005;91:334–338.
116.WladimiroffJW,HuismanTW,StewartPA.Fetal
cardiacflowvelocitiesinthelate1sttrimesterof
pregnancy:atransvaginaldopplerstudy.JAm
CollCardiol.1991;17:1357–1359.
117.WladimiroffJW,StewartPA,BurghouwtMT,
StijnenT.Normalfetalcardiacflowvelocity
waveformsbetween11and16weeksof
gestation.AmJObstetGynecol.1992;167:736–
739.
118.vanderMoorenK,BarendregtLG,Wladimiroff
JW.Fetalatrioventricularandoutflowtractflow
velocitywaveformsduringnormalsecondhalf
ofpregnancy.AmJObstetGynecol.
1991;165:668–674.
119.vanSplunderIP,StijnenT,WladimiroffJW.
Fetalatrioventricular,venousandarterialflow
velocitywaveformsinthesmallforgestational
agefetus.PediatrRes.1997;42:765–775.



×