546
KHO SÁT RI LON NHP VÀ THI
HUYNG BNG HOLTER ECG 24 GI
Dung, Trn Minh Trí
1
, Hu
2
1
2
M: hii lon nhp vi ng dn tim 24 gi trong
lâm sàng, chúng tôi tin hành nghiên c tài vi các mc tiêu sau: (1) Kho sát t l ri lon
nhp, thiu máu cc b n tim 24 gi; nh t l, tn sut ri lon
nhp trên Holter theo nh
ng nghiên cu: Bt áp vào khoa tim mch bnh vin N guyn
Trãi t n tim 12 chuyng.
cu: Nghiên cu ct ngang tin cu mô t.
Kt qu: (1) T l các ngoi tâm thu tht, ngoi tâm thu trên thi
c phát hin trên holter 24 gi khá cao. (2) T l các ri lon nhp phc tp, nguy him
(ngoi tâm thu tht côi, nhanh th
ban ngày. (3) Huyt áp càng cao th t l xy ra ri lon nhp và thiu máu máu cc b
càng nhiu.
Kt lun: Nghiên cu nh giúp phát hin nhiu ri lon nh và s
giúp s dng thuu tr tt áp.
T V
-
-
Tiêu chuẩn nhận bệnh: 123 b t áp vào khoa tim mch 2 bnh vin
Nguyn Trãi t n tim tng
Tiêu chuẩn loại trừ: Các bnh mang tính cht cp tính (Bnh nhân nhp, viêm
màng ngoài tim cp, tai bin mch máu não ct cp suy thn mn, xut huy
- ng ý mang máy, mang máy b rn cc,thi 20 gi.
[3]
-
-
-
547
-
-
-
-
gian
t áp: theo khuyn cáo ca hi Tim mch Vit Nam.2006-2010
i
Chung
51.20±7.69 (56)
73.25±8.80 (67)
63.21±13.78 (123)
Nam, n(%)
33/56 (58.93%)
27/67 (40.30%)
60/123 (48.78%)
NTTTrT >100/24G§
NTTT>100/24G§
TMCT§§
23 (18.7%)
22 (17.9%)
33 (26.8%)
18 (14.6%)
6 (4.9%)
6 (4.9%)
25 (20.3%)
7 (5.7%)
Nam
15 (12.2%)
13 (10.6%)
38 (30.9%)
10 (8.1%)
14 (11.4%)
15 (12.2%)
20 (16.3%)
15 (12.2%)
11 (8.9%)
6 (4.9%)
15 (12.2%)
7 (5.7%)
18 (14.6%)
22 (17.9%)
43 (35.0%)
18 (14.6%)
-
-
-
-
Ban ngày 6g 20g
6g
17/28 (60.7%)
17/28 (60.7%)
11/28 (39.3%)
4/28 (14.3%)
3/28 (10.7%)
3/28 (10.7%)
3/28 (10.7%)
3/28 (10.7%)
3/28 (10.7%)
13/28 (46.4%)
13/28 (46.4%)
11/28 (39.3%)
16/28 (57.1%)
16/28 (57.1%)
13/28 (46.4%)
-
Ban ngày 6g 20g
6g
5 (29.4%)
12 (70.6%)
3 (27.3%)
8 (72.7%)
0 (0%)
3 (100%)
0 (0%)
3 (100%)
0 (0%)
3 (100%)
0 (0%)
3 (100%)
2 (15.4%)
11 (84.6%)
2 (18.2%)
9 (81.8%)
548
4 (25.0%)
12 (75.0%)
4 (30.8%)
9 (69.2%)
-
Ban ngày 6g 20g
6g
6/25 (24.0%)
3/25 (12.0%)
10/25 (40.0%)
14/25 (56.0%)
Hol
3],
2].
- 5
12
lâm sàng.
1][2], Hirofumi Tasaki [6
9]. Các nghiên
sáng [10
8
8
7
11],
Nhóm c
y.
549
1.
-40.
-175.
2.
, tr
12-23.
3.
4.
; tr 278-282.
5. C. Michael Gibson et al (2007). Diagnostic and prognostic value of ambulatory ECG
(Holter) monitoring in patients with coronary heart disease: a review. Journal of
Thrombosis and Thrombolysis.135-145.
6. Hirofumi Tasaki et al (2006). Longitudinal Age-Related Changes in 24-Hour Total Heart
Beats and Premature Beats and Their Relationship in Healthy Elderly Subjects. nt Heart
J;47: 549-563.
7. Kannel WB, Benjamin EJ (2008). Status of the epidemiology of atrial fbrillation.
Med Clin North Am; 92(1):1740, ix.
8. Mark A. Wooda et al (1995). Circadian pattern of ventricular tachyarrhythmias in
patients with implantable cardioverter-defibrillators. Journal of the American College of
Cardiology; Volume 25, Issue 4, Pages 901-907.
9. Messerli FH et al(1984). Hypertension and sudden death. Increased ventricular ectopic
activity in left ventricular hypertrophy. Am J Med ; 77: 18-22.
10. Seigel D, Black DM, Seeley DG et al (1992). Circadian variation in ventricular
arrhythmias in hypertensive men. Am J Cardiol; 69:344-347.
11. Yoshiaki Deguchi, Mari Amino et al (2009). Circadian Distribution of Paroxysmal Atrial
Fibrillation in Patients with and without Structural Heart Disease in Untreated State.
Annals of Noninvasive Electrocardiology; Volume 14 Issue 3, Pages 280 289.
12. Zehender M, Meinertz T, Hohnloser S, Geibel A (1992). Prevalence of circadian
variations and spontaneous variability of cardiac disorders and ECG changes suggestive
of myocardial ischaemia in systemic arterial hypertension.Circulation; 85: 1808